カズレーザーと学ぶ。2024年12月31日 年末年始に家族や親せきと知りたい!血液型and県民性の新常識SP
#EnglishMovie #cdrama #drama #engsub #chinesedramaengsub #movieshortfull
#EnglishMovie #cdrama #drama #engsub #chinesedramaengsub #movieshortfull
Category
📺
TVTranscript
00:00It's a little difficult, but if you know the new knowledge that changes your life, you can learn a lot!
00:08The latest science special that will make you look good!
00:11If your tongue falls, it will become a double chin.
00:14It's a way to get rid of the swelling in your face in 5 minutes.
00:18There are many ways to get rid of the swelling that can be done right now!
00:21The premise so far has been quite overturned.
00:24In addition!
00:25It's a way to make you live longer!
00:30What is the food that makes you live longer in recent years?
00:33I'm surprised!
00:35Kazuto Learns Tuesday Night Kuji!
00:38Next year, on January 14th, Tuesday Night Kuji will be broadcast!
00:42Today on New Year's Day, I want to know my family and relatives at the end of the year!
00:47Kazuto Learns the New Knowledge of Blood Type and Genitalia!
00:50Kazuto Learns the New Knowledge of Blood Type and Genitalia!
00:55There's a lot to talk about, isn't there?
00:58Kazuto, you don't believe in blood type fortune-telling, do you?
01:03I don't believe in it.
01:05How about you, Muto?
01:07I really like fortune-telling, so I don't get along well with type B and type A.
01:11I also love blood type, so I'm worried about it.
01:14I'm worried about it.
01:16There's something in the corner of my mind.
01:19I'm worried about type A.
01:22Here are today's key points.
01:25First, there is a regional difference in obesity.
01:29It was found in the latest genome analysis.
01:32Obesity?
01:34Does it affect your body type?
01:37Second, be careful not to get hurt with type A.
01:40There is a risk of illness for each blood type.
01:41Be careful not to get hurt with type A.
01:47I don't believe in fortune-telling.
01:52I think there was a suspicious rumor that there are many types of blood in Asia.
02:02Which one should we start with?
02:04Let's start with blood type.
02:06Type A is a risk of illness for each blood type.
02:09This is Professor Koichi Fukase, a professor of science at the University of Osaka.
02:15Nice to meet you.
02:18I've been hearing a lot about blood type.
02:21Is it related to fortune-telling?
02:23I think it's related to fortune-telling.
02:26I'm sorry to say this at the beginning,
02:28but there is no scientific basis for the division of blood types.
02:34Thank you for making it clear.
02:36However, we've come to know that there is a difference in the risk of certain diseases depending on the blood type.
02:43I didn't know that.
02:45I see.
02:47Is this true?
02:49It's true according to statistics.
02:52People who don't have blood type tend to say that statistics are correct.
02:56Today, we're going to talk about why the risk of illness differs depending on the blood type.
03:05Professor Koichi Fukase, a professor of science at the University of Osaka.
03:13In fact, there are different types of diseases depending on the blood type.
03:19From type B, which is vulnerable to infectious diseases,
03:22to type C, which is hard to stop bleeding when injured,
03:25to type D, which is high risk of stroke and cardiac arrest.
03:29We're going to learn a lot about the new knowledge of blood types.
03:39There are 40 types of blood types.
03:42The most famous one is type AB.
03:47By the way, Mr. Oda, do you know what's the difference between type AB and type O?
03:51I'll tell you.
03:53Type AB is A, B, and O, right?
03:56What's the difference?
03:57What's the difference?
03:59What's he doing?
04:01He's provoking us.
04:03He's a great teacher.
04:05He's a math teacher.
04:07I thought there must be a difference.
04:09It's like the content.
04:11The content?
04:13The content?
04:15The content is different!
04:17You can't do anything with a loud voice.
04:19Mr. Kazu, do you know?
04:20Yes, I do.
04:22I found it in a textbook.
04:24I found it in a textbook.
04:26That's great.
04:28I think it's the difference between the content and the source.
04:30I think it's the difference between the content and the source.
04:33In the old days, blood was sucked into the bloodstream until you found it.
04:37There were people who died even if they had blood in their bloodstream.
04:40No way!
04:42I read this in an electric book.
04:44My father is type B, my mother is type AB, and I'm type BB.
04:47There are people who are type BO.
04:50How is that different?
04:52It's different in terms of genes, but there's no difference in terms of blood type.
04:57Are type BO and type BB the same?
04:59It's the same.
05:01By the way, is there a blood type other than type A, B, O?
05:05In medical dramas, it's RH plus or minus.
05:08I've heard of it.
05:10People who are type AB and type RH plus or minus have low blood type, so they often say that they don't have enough blood.
05:15It's the rarest.
05:17Japanese people have a 0.5% rate of RH plus or minus.
05:21There are 200 people, so type AB and type RH plus or minus are very rare.
05:28From the phenomenon that hardens when different blood types are mixed,
05:32a blood type was discovered by Austrian Rantz Steiner in 1900.
05:37Due to a certain difference in blood type,
05:40it is divided into four familiar types, but I will explain it later.
05:44Why was type AB blood type, which was only medical knowledge, used only in Japan for fortune-telling?
05:54There was a theory before the war, but there was a book published about 50 years ago.
05:59It is a book called Aisho, which can be understood by blood type.
06:02It is a book called Aisho, which can be understood by blood type.
06:05It is a book called Aisho, which can be understood by blood type.
06:09It started with this book?
06:11Yes.
06:12Type B is my pace, and type A is the basic.
06:15Many people still believe in this blood type, but this book is the beginning.
06:20Wow.
06:23The reason why this happened is that Japanese blood types are evenly distributed between type A, B, AB, and type O.
06:31That's why it became popular.
06:34It's different from other countries.
06:36Type A is 40%, type O is 30%, type B is 20%.
06:39It's easy to understand.
06:41Why is it so evenly distributed?
06:44This percentage is relatively high in South Korea, China, and East Asia.
06:48It's different from other countries.
06:50Type A was first separated from chimpanzees and monkeys.
06:54Type B was separated about 3.5 million years ago.
06:57After that, type O was also separated from type A.
07:01Type A was the first?
07:03Yes, type A was the first.
07:05I see.
07:06I heard that Venezuelans and Colombians have large blood types.
07:09It's almost 100%.
07:11Really?
07:13It is said that most of the people who came to the United States from the Venetian were large.
07:19I heard that Venezuelans have large blood types.
07:22I heard that Venezuelans have large blood types.
07:24I heard that Venezuelans have large blood types.
07:26I see.
07:28We have learned that there are many types of blood in recent years from various studies.
07:34This is related to certain diseases.
07:38Type A and B are the weakest.
07:40Why?
07:42We have learned that there are many types of blood in recent years from various studies.
07:47This is related to certain diseases.
07:52There are many regions with large blood types in the world.
07:59Japan is a rare environment where blood types are evenly distributed.
08:04It is thought that blood type fortune-telling has become popular.
08:07Then, why is the probability of getting sick different depending on blood type?
08:13The big point is the difference in the probability of getting sick depending on blood type.
08:18What does that mean?
08:21The light source is a marker that marks the cells.
08:25In addition, antibodies are made to eliminate the disease when it enters the body.
08:32The light source and the antibodies are perfect for the relationship between the key and the keyhole.
08:36It is recognized as a foreign object and is attacked and eliminated.
08:40It is attacked in the same way as the shape you are in charge of.
08:42What does that mean?
08:44The antibodies of blood type B attack the light source of blood type A.
08:50As a result, blood type B is attacked by blood type A.
08:55Blood type B becomes hard.
08:57Blood type B is attacked by blood type A because it is a foreign object.
09:03That's right.
09:05In addition, the blood type mark is not only on the surface of the blood vessels.
09:10It is on all the cells in the body.
09:14It's not just blood.
09:17It is on the skin, the cells in the intestines, and the body fluids.
09:21It's on the nails, too.
09:23There are few hair on the nails, but there are a lot of hair on the hair.
09:28I see.
09:30There is an immune system that eliminates foreign objects in our body.
09:34The light source is the foreign object for the body.
09:37The substance that eliminates the light source is called the antibody.
09:40Antibodies have the property of attacking only a specific light source.
09:45And this light source is said to cause a difference in diseases that are easy to get in each blood type.
09:51Why does this difference in light source affect the ease of disease?
09:56That's because the blood type mark light source is different, which is easy to get infected by viruses.
10:02I see.
10:04So, the disease that you aim for determines the ease of infection?
10:12Yes, it changes.
10:14I see.
10:16This virus is easy to get in the light source of the circle, but it is difficult to get in the light source of the Y-shaped blood vessel.
10:22This changes the degree of infection.
10:25For example, if a virus that is easy to get in the light source of the blood vessel of type A enters the blood vessel of type A,
10:28it is easy to get infected by the virus in the blood vessel of the other blood type.
10:33Is there a possibility that a virus that is easy to get infected by the virus of type A will infect a person of type B?
10:38Of course, it is a probability problem.
10:41Even if the light source does not fit, if it simply gets in a lot, it is likely that it will enter the cells of other people.
10:48I see.
10:50When a virus is infected, it is often the case that the body cells target this light source.
10:56Some viruses are more susceptible to A-type viruses, while others are more susceptible to B-type viruses.
11:05As a result, blood types are different for each disease.
11:10By the way, A and B viruses are more susceptible to A-type viruses and B-type viruses.
11:15Therefore, A-type viruses and B-type viruses are more susceptible to A-type viruses.
11:23Another issue is that blood types are more susceptible to A-type viruses, while others are more susceptible to B-type viruses.
11:31There are many differences in blood types between the two diseases.
11:35In fact, blood types are different for each disease.
11:40Oh, I see.
11:43So blood types are different for each disease.
11:45Then, is it that blood type A is less suspicious of B-type viruses?
11:47Blood types A and B are less suspicious of B-type viruses.
11:51The blood type is A, which is easy to solidify.
11:53The blood type of large blood types is B, which solidifies blood when injured.
11:58However, the blood type of large blood types is B, which is twice as high as the blood type of small blood types.
12:01Therefore, the mortality rate due to injury is more than twice as high as that of other blood types.
12:07Then, what effect does the blood type of large blood types have on the ease of blood to solidify and the difference between the blood type of large and small blood types?
12:17Based on this, I would like to announce the diseases to be careful about for each blood type.
12:23First of all, Mr. Tsukasa, please announce the diseases that people with blood type AB should be careful about.
12:29From now on, we will announce the diseases to be careful about for each blood type.
12:33First of all, let's start with the 10% of Japanese people with blood type AB.
12:38The diseases that people with blood type AB should be careful about are
12:43stroke and myocardial infarction.
12:48Stroke and myocardial infarction?
12:50Yes.
12:51Why?
12:53People with blood type AB have 1.83 times more risk of stroke than people with large blood types.
12:58Myocardial infarction risk is 1.82 times higher.
13:02So why?
13:04Why?
13:06I don't know for sure.
13:09Compared to myocardial infarction, people with blood type AB have the lowest risk of myocardial infarction.
13:13Is that so?
13:15Myocardial infarction and myocardium infarction are inseparable.
13:19Myocardial infarction affects myocardium infarction.
13:24This is a scientific fact.
13:29Myocardial infarction affects myocardium infarction.
13:40People with blood type AB has low immune system.
13:43It is related to myocardial infarction.
13:47People with blood type AB have high risk of stroke and myocardium infarction.
13:51Like I said, people with blood type AB and people with blood type B are at risk for stroke.
13:57risk, so please be careful about what you say next.
14:01Next, what is a disease that type B should be careful about?
14:05Type B, among them, Mr. Buto.
14:10The disease that type B should be careful about is
14:15diabetes.
14:19It is known that the incidence of type B diabetes is high in the follow-up investigation aimed at French women.
14:27What is being said now is that type B blood clots easily, which increases the rate of blood clotting, but it is said that this is one of the causes of insulin resistance.
14:39In addition, I think that the recent imbalance in blood vessels may cause inflammation and reduce insulin.
14:46Earlier, you said that type O blood clots easily, right?
14:51Does that mean that type O people are less likely to get diabetes?
14:54That's right.
14:56I'm type O, but I was told that I had a hidden diabetes.
14:58That's the most dangerous.
15:00It depends on the person.
15:02It depends on the person.
15:04If you take it big, there is also such a direction.
15:06About 90% of the people in my family are type B and have diabetes.
15:12So I thought it was the same.
15:14I felt like I had fallen this time that I was type A.
15:18If you are careful about it.
15:20If you are careful from now on, you can change it depending on your lifestyle, right?
15:26Yes, that's right.
15:28In addition, there is a tendency that type B is also easy to be infected with other diseases.
15:34What are the diseases that type B should be careful about?
15:40We will announce the diseases that are easy to be infected by blood type.
15:44According to Dr. Fukase of Osaka University, type B is more likely to have diabetes than other blood types.
15:52In addition.
15:54In addition, type B is also easy to be infected with pneumonia and blood clots.
16:00It's not serious.
16:02It's not serious.
16:04It used to be an incurable disease.
16:06Why is that?
16:08I don't know the reason for this yet.
16:10Our immune system has a risk of missing pneumonia and blood clots compared to other blood types.
16:18On the contrary, is there anything that type B is strong against this?
16:22There is data that type B is difficult to become anemic.
16:26Anemic?
16:28Anemic?
16:30Is that right?
16:32I don't think it's right.
16:34I don't think it's right.
16:36I don't think it's right.
16:38I don't think it's right.
16:40I don't know the specific mechanism, but type B is more likely to have blood clots than type A.
16:46Inflammation is more likely to occur.
16:54Next is type O.
16:56Next is type O.
16:58There is a risk of anemia that type O is difficult to become anemic.
17:02Mr. Kubozuka, Mr. Yada, and Mr. Kazu.
17:06Please announce the disease that type O should be careful about.
17:12The diseases that type O should be careful about are
17:16type O and type B.
17:20Why are there two diseases?
17:22I found that type O is about 1.3 times more likely to become anemic than other types of blood.
17:30That's a lot.
17:32Why is it easy for type O to become anemic?
17:34It is because of the strength of the combination of pyrrolecine and type O.
17:40Is it okay if there is no pyrrolecine?
17:42If you take pyrrolecine?
17:44If you take pyrrolecine, it's okay.
17:46It's the same mechanism as type O.
17:48It is easy for type O and type A to become anemic.
17:50It is easy for type O to become anemic.
17:52I hate it.
17:54I like raw oysters.
17:56Oysters are the curse of the virus.
17:58Oysters are the curse of the virus.
18:00I got it right.
18:02I don't like oysters either.
18:04I got it right four times.
18:06I like raw oysters.
18:08You got it right three times, but you still got it wrong four times.
18:10I tried it four times.
18:12I tried it four times.
18:14I thought I would quit if I got it right.
18:16I got it right.
18:18So I swore I wouldn't eat it for the rest of my life.
18:20As a result of research in China,
18:22it is 1.28 times more infectious than the curse virus.
18:24it is 1.28 times more infectious than the curse virus.
18:26It's a curse virus,
18:28but it's not as infectious as type B.
18:30Type B is a little better.
18:32Type B is a little better.
18:34I'm glad.
18:36What about type O and other diseases?
18:38What about type O and other diseases?
18:40Type O157, typhoid fever, etc.
18:42Type O157, typhoid fever, etc.
18:44It's easy to get small-blood type diseases.
18:46It's easy to get small-blood type diseases.
18:48It's easy to get small-blood type diseases.
18:50How do you feel?
18:52That's not what he says.
18:54I'm really weak in my stomach.
18:56I'm really weak in my stomach.
18:58That's not all the reason.
19:00I think so.
19:02I think so.
19:04I think so.
19:06According to theŞōkōōnō-san,
19:08It's easy for viruses that cause digestive diseases, such as colobirus and colorectal cancer, to get together, so you need to be careful about what you eat.
19:17By the way, O-gata has a surprising result in addition to his illness.
19:22By the way, what kind of personality does O-gata have?
19:26He's very bold.
19:28He doesn't care.
19:30He's calm.
19:32This is a study from the University of East Tennessee in the United States.
19:35This is a comparison between O-gata and A-gata.
19:38O-gata is less stressed than A-gata.
19:42Oh, I see.
19:45I thought it was the opposite.
19:48If digestive diseases are affected by O-gata,
19:51it's because of cortisol.
19:55O-gata has a lower level of cortisol than A-gata.
20:01It's slow to recover from stress.
20:04That's the report.
20:07Is there a disease that is difficult for O-gata to get?
20:11O-gata is difficult to get high blood pressure.
20:14High blood pressure.
20:16He's not good at writing.
20:19It's difficult for blood to solidify.
20:23It's difficult for O-gata to get high blood pressure.
20:26It's difficult for O-gata to get high blood pressure.
20:29I see.
20:31Finally, what is the disease that A-gata should pay attention to?
20:37A-gata should pay attention to this.
20:40Oda-san and I will ask O-gata.
20:43Please.
20:45The disease that A-gata should pay attention to is...
20:49A-gata has high blood pressure and high blood pressure.
20:51It's a serious problem.
20:54It's a serious problem.
20:56Don't speak so loudly.
20:58It's a serious problem.
21:00It's not related to A-gata.
21:03It's a little scary.
21:05It's a little heavy.
21:08A-gata has 1.25 times higher risk of high blood pressure and high blood pressure than other blood types.
21:16A-gata has 1.79 times higher risk of high blood pressure and high blood pressure than other blood types.
21:21It's very high.
21:23Why? What kind of mechanism is it?
21:25It's easy for blood to solidify.
21:28It's easy to get blood clots.
21:30It's easy to get blood clots.
21:32It's easy to get blood clots.
21:35What's so funny?
21:37It's not funny.
21:39So, in that sense, A-gata is good for blood clots?
21:45Yes, it is.
21:47It's good for blood clots.
21:49It's good for the brain.
21:51It's good for the brain.
21:53It's good for the brain.
21:55It's good for the brain.
21:57It's good for the brain.
21:59Why is A-gata more dangerous than B-gata?
22:04Compared to A-gata, B-gata has a higher risk of blood clots.
22:09But I don't know why.
22:14You don't know, but A-gata is more dangerous.
22:16A-gata has a higher risk of blood clots.
22:19What about A-gata?
22:22It's easy to get blood clots.
22:25It's easy to get blood clots.
22:28Zero?
22:29There's no merit.
22:31There's no merit.
22:33That's all?
22:35But why is there such a big difference between blood types?
22:42In the first place, why did humans evolve with a variety of blood types?
22:48It's because of the world's blood type distribution.
22:53The relationship between blood types and diseases is shown in the blood type distribution.
22:58There are many A-gata in Africa.
23:00In fact, malaria is more likely to infect A-gata.
23:07That's why there are many A-gata with strong malaria in Africa.
23:12There are many A-gata in Europe.
23:15These are more likely to infect A-gata.
23:18There's such a thing?
23:20In the 19th century, there were more A-gata than A-gata.
23:24In the 19th century?
23:25It's so recent.
23:26It's close to natural disease.
23:29The distribution of blood types is to prevent infections between pathogens and humans.
23:35As a result of the evolution of bio-defense function,
23:39we know that the blood type system is a risk hedge to avoid the extinction of species.
23:46If there's a single A-gata, it's a big sacrifice.
23:52So it's scattered in the first place?
23:54That's right.
23:55It's a mechanism to prevent the spread of the virus by scattering it.
24:01Is it a distribution of blood types to survive?
24:05Yes.
24:06In particular, it is clear that there is a trend of new coronavirus,
24:11and A-gata is the strongest and hardest to infect.
24:15What is the blood type that is the hardest to infect by coronavirus?
24:21There is a difference in the risk of disease that is easy to be infected by blood type,
24:26but there is a new research that has been paying attention in recent years.
24:31There is a trend of new coronavirus,
24:34and A-gata and blood type are the strongest and hardest to infect.
24:40Oh, really?
24:41Yes.
24:42I didn't know that.
24:44Compared to A-gata and other A-gata,
24:47there is a difference in the risk of disease that is easy to be infected by blood type,
24:50but there is a difference in the risk of disease that is easy to be infected by blood type.
24:55Why is the risk of infection low only for A-gata people?
25:01Let's find out why.
25:06Actually, it's a virus that infects humans,
25:09and we know that the virus transforms into the blood type of the infected person.
25:15Oh, really?
25:18The virus enters the human body and increases itself,
25:21so it takes away the mold of the base?
25:24That's right.
25:26That's amazing.
25:27The virus increases in the human cell,
25:30but when it leaves the cell,
25:32it also takes away the blood type that exists in the cell membrane.
25:37That's why A-gata people have A-gata virus,
25:41B-gata people have B-gata virus,
25:43and O-gata people have O-gata virus.
25:45Oh, I see.
25:47So, if I catch a cold with B-gata virus,
25:50the B-gata bacteria that has the antigen comes out,
25:55and it's easy to infect B-gata people?
25:58Yes, it's easy to infect B-gata people.
26:00But it's hard to infect A-gata people from B-gata people.
26:04And it's hard to infect O-gata people from B-gata people.
26:06So, O-gata people have antibodies, so it's hard to infect them?
26:10Yes.
26:11O-gata people can be infected from A-gata people, B-gata people, and A-B-gata people.
26:15I see.
26:16So, O-gata people are hard to infect by coronavirus?
26:19Yes.
26:20So, O-gata people...
26:22First of all, viruses are all in the same system,
26:25so they are strong against infectious diseases.
26:28Yes.
26:29But O-gata people are hard to infect.
26:32Oh, so it's hard to infect O-gata people?
26:34I see.
26:35Because they spread the virus of Mujirushi?
26:38Yes.
26:39I see.
26:40And A-B-gata people are hard to infect by everyone.
26:43I see.
26:44It's hard to infect everyone.
26:46But is it hard to infect many people from A-B-gata people?
26:49It's easy to infect A-B-gata people from A-B-gata people,
26:52but it's hard to infect everyone else.
26:54I see.
26:55When a coronavirus infects a person,
26:58it has an antigen of the blood type that grows in the cell.
27:02Therefore, viruses that infect A-gata people
27:05are easy to infect A-gata and A-B-gata people,
27:09but B-gata and O-gata people with antibodies to A-gata are hard to infect.
27:15In particular, O-gata people have antibodies to both A-gata and B-gata,
27:20so they are hard to infect by viruses.
27:24Can we say the same thing about viruses other than coronaviruses?
27:28That's right.
27:29It's the same for colds and influenza viruses.
27:32Viruses that have a cell membrane are all of this type.
27:36I see.
27:37I thought it was a joke,
27:39but when I analyzed it and explained it,
27:42I was very satisfied.
27:46There was evidence.
27:48Did you get infected by coronavirus?
27:50No, I didn't.
27:51But I get infected by influenza every year.
27:54It's the best.
27:55It's easy to get infected.
27:56It's hard to get infected.
27:57I can't say I don't get infected.
27:58I hope everyone will understand the characteristics of A-gata
28:02and connect it to the prevention of illness.
28:05I don't think A-gata fortune-telling will disappear.
28:08Everyone likes it.
28:09But after talking about A-gata fortune-telling,
28:11I want you to tell me everything about today.
28:14It was a great video.
28:16Let's move on to the next topic.
28:19Fatness and lightness are distinguished by regionality.
28:21It is a common feature found in the latest genome analysis.
28:24This is Professor Ohta Hiroki,
28:26a professor of biology and chemistry at Tokyo University.
28:30Nice to meet you.
28:31Nice to meet you.
28:33Can you look at this map?
28:35It's a map of Japan.
28:37What kind of map do you think this is?
28:41It's very dark in Hokkaido.
28:43It's dark in Kyushu, too.
28:45It's really dark in Saitama.
28:47Is it true?
28:49It's because of the strong alcohol.
28:51The strong alcohol?
28:53I think it's true.
28:55It's also true in Kyushu.
28:57It's also true in Hokkaido.
28:59It's also true in Hokkaido?
29:01We've just got the answer.
29:03It's because of the strong alcohol.
29:06It's very distinguished by regionality.
29:08It's because of the strong alcohol.
29:10To be exact,
29:11the people who have a weak alcohol tolerance
29:14are in the lower class and often go to Kansai.
29:16Kansai is weak.
29:19Let's look at another map.
29:21The Tohoku region is dark again.
29:23It's completely different.
29:24This is also the frequency of genetic mutations in a certain body type.
29:30But Okinawa and Niigata are still dark.
29:33It's easy to get sick.
29:35Ah, sickness.
29:37In fact, there are many prefectures with mutations that are easy to accumulate neutrophils.
29:44Why is there such a difference?
29:47It's related to the difference in body type.
29:51It's a great story of how people who live in Japan have been formed.
29:59There was a great story that the body type of modern Japanese people goes back to the Hyōga period.
30:06What is the difference in disease that the region should be careful of?
30:11Professor Hiroki Ōta, a graduate student at Tokyo University.
30:19Even in Japan, there is a similar body type in the Tohoku and Kyushu regions of Hokkaido.
30:26In the Kansai and China regions, there is a reverse trend.
30:30Why is there such a difference in the body type of genes in the region?
30:37Based on the reason, we can see the great history from the Hyōga period to the birth of modern Japanese people.
30:45Learn the old and new civilizations that modern Japanese people should know.
30:52Why is there such a difference in the region?
30:55I'd like you to look at this illustration.
30:57Do you remember this?
30:59In the textbook.
31:01There was something like Jōmonjin, Yayoijin.
31:04Jōmonjin and Yayoijin are two well-known groups of ancestors of people living in Japan.
31:13Modern Japanese people were born from the confluence of these two groups.
31:19About 10 to 20 years ago,
31:22the technology to examine DNA came from the bones that came out of the remains.
31:28A lot of things have come to light.
31:30About 300,000 years ago, Homo sapiens spread to Africa.
31:35How did they get to Japan?
31:38What route did they follow from Africa?
31:40After they left Africa.
31:43Jōmonjin is a world heritage site.
31:46Hokkaido is in the north of Tohoku Prefecture.
31:49The most famous Yayoijin ruins are in Saga Prefecture and Yoshinogari Ruins.
31:54I think that's how much remains are left.
31:58I think it came from the north.
32:01As Mr. Kazuma said,
32:03it's very similar to the ruins of Baikal Lake found in Japan.
32:09So I think it came from the north in archaeology.
32:13But I've heard that the shape of the bones is similar.
32:18It was a mystery.
32:21The culture of the Jōmon period was in Siberia.
32:24The shape of the bones was similar to that of Southeast Asia,
32:27so the roots of the Jōmon people have been shrouded in mystery for a long time.
32:32So Mr. Oza's group compared the bones found in the ruins of Southeast Asia
32:37with the genetic information of the bones of the Jōmon people.
32:41It turned out to be quite similar to the ancient genetics of the people living around Naos.
32:48As a result, the roots of the Jōmon people have been shrouded in mystery for a long time.
32:53Apparently, the ancestors of the Jōmon people came from the south.
32:59There are a minority of people called Mani people in Thailand.
33:02They live in the jungle near the border between Malaysia and Thailand.
33:05They are still the main population.
33:08Are these people the closest to the Jōmon people?
33:12The ancestors of these people and the ancestors of the Jōmon people had a common ancestor.
33:17So it's a little different to say that the Jōmon people were these people.
33:21It's close to the ancestors, so it's likely that they came from that route.
33:26At that time, they moved from Southeast Asia to Japan, which was connected to the continent.
33:31After that, the sea level rose, separating Japan from the continent.
33:36The Jōmon people, the ancestors of the current Japanese people, were left there.
33:41After that, the current Japanese people were born, crossing the sea from the continent.
33:47For this reason, the Japanese people are still happily inheriting the genes of the Jōmon people.
33:54You all have about 25,000 genes.
33:58The part inherited from the Jōmon people is about 10% to 20% on average.
34:04That's a lot.
34:06That's a lot.
34:07But 10% to 20%?
34:09The number of genes inherited from the Jōmon people varies from person to person.
34:13I think so, too.
34:14If you count how many genes you have, you can see the Jōmon people.
34:19I see.
34:20In addition, we found that there is a clear difference in the area of the Jōmon people.
34:29The darker the color, the higher the Jōmon people.
34:32There are individual differences, but if you look at it by prefecture, it looks like this.
34:37There are more Jōmon people in the north.
34:39The north is darker overall.
34:42But if that's the case, I can't help but remember that the Jōmon people came from the north.
34:47But Okinawa is also dark.
34:48Yes, Okinawa is also dark.
34:50The reason why there is no Hokkaido now is that it is a place where people emigrated after the Meiji period, so it is difficult to investigate.
34:58The light areas are around Kansai.
35:02Yes, Kansai and Shikoku.
35:04This area is a place where the population increased from the Jōmon period to the Yayoi period.
35:10I see.
35:11As you can see from the number of ruins, the number of Jōmon people has decreased in the area where people came in from the mainland.
35:20Is Kansai closer to Kyushu than the mainland?
35:24I don't know about this area yet.
35:28It is said that the genetic variation from the Jōmon people has some effect on the quality of the body.
35:41Is there a difference in the risk of body quality and illness in areas with high Jōmon people and areas with low Jōmon people?
35:49Is there a difference in the risk of body quality and illness in areas with high Jōmon people?
35:54It is almost decided by one gene.
36:00The genetic variation from the Jōmon people has some effect on the quality of the body.
36:09The Jōmon people's identity is clearly seen by the local people.
36:12In fact, this created a difference in the quality of the body in the area.
36:18The strength of the alcohol introduced earlier is also due to the influence of the Jōmon people's genes.
36:24When you drink alcohol, the ethanol is decomposed in the liver and turns into acetaldehyde.
36:31Acetaldehyde makes you feel sick or have a headache.
36:36The genetic variation from the Jōmon people has some effect on the quality of the body.
36:40People who have the gene from the Jōmon people can't drink alcohol.
36:44Both people can drink alcohol with the gene from the Jōmon people.
36:49People who have the gene from the Jōmon people are weak.
36:53People who have the gene from the Jōmon people cannot drink alcohol.
36:55They are highly alcoholic.
36:57It is almost decided by one gene.
37:00I've been talking about the quality of the body.
37:03Basically, the quality of the body is not decided by one gene.
37:07For example, the fact that Mr. Kurosawa is tall is related to more than 100 genes.
37:13But this is very clear.
37:17It is decided by whether you have this gene or not.
37:20It's rare.
37:22Were the people who were born in Africa strong?
37:30That's a very important question.
37:32They could drink alcohol.
37:35This gene was born in East Asia.
37:41Most of the genes that humans have are in Africa.
37:46This gene was born in East Asia.
37:50Interesting.
37:51Genes that are weak in alcohol are concentrated in the lower part of Japan.
37:58The reason why there are many people who are weak in alcohol in the Kansai and China regions
38:02is probably because many people have moved from the mainland to this region.
38:08And the influence of the Jōmon people is not only in the quality of the body.
38:13It depends on whether you are fat or not.
38:16Is that so?
38:19Most of our bodies have many genes that can cause diseases.
38:25If you look at the relationship between the many genes and the way our bodies behave,
38:29the people who have the genetic mutation of the Jōmon people
38:32have hemoglobin A1c, which indicates high blood sugar levels,
38:36and neutral fatty acids.
38:40Was it easy for the Jōmon people to get fat?
38:44It was easy for the Jōmon people to get fat if they lived the way they do now.
38:50Does that mean the higher the Jōmon people are,
38:53the more likely they are to have this mutation?
38:56The quality of the body that is easy to get fat is not determined by one gene.
39:00I'm talking about one of them now.
39:03By the way, if you look at the height of the BMI, which indicates the level of obesity,
39:08especially in women, it is in sync with the map of the Jōmon people,
39:12and the Tohoku people are at a higher level.
39:15Does that mean the Tohoku people don't like sweets?
39:19I don't think it has anything to do with their lifestyle,
39:23but in order to eliminate the impact of their lifestyle,
39:27we conducted a study on the obesity rate at the age of five.
39:31Five years old?
39:33What about the Jōmon people?
39:35The high obesity rate at the age of five
39:38and the high Jōmon population are relatively similar.
39:44Why are the Jōmon people easy to get fat?
39:48The Jōmon people have been collecting feed,
39:51so you can imagine it from here,
39:54but I think it was hard to get food.
39:57Especially in the Soe period, the Hyōga period,
40:00there was very little food,
40:02so it was easier to survive for those who were in good shape.
40:06I see, so they were able to survive on a little food.
40:10They were trying to keep fit.
40:13This is called a contractual gene.
40:16After the Hyōga period, the Jōmon people were trapped in the Jōmon network,
40:21so they continued to collect feed.
40:24But compared to that, agriculture started on the mainland.
40:27In the Soe period, the Jōmon people were able to get a lot of carbohydrates.
40:32In other words, the Jōmon people were able to get a lot of carbohydrates.
40:36This is what you see in this picture.
40:39If you think about the history of mankind,
40:42it was overwhelmingly advantageous to accumulate food for a long time,
40:46but suddenly civilization developed,
40:48and everyone started to make rice paddies,
40:51and suddenly it turned into a bad image of getting fat.
40:54It's a strange story that what the ancestors of the ancestors got
40:57is now a target of bad words.
40:59That's right.
41:00Is there a disease that is easy for Torai people to get?
41:03On the contrary, people with low Jōmon levels
41:06are more likely to have allergies and genetic mutations.
41:16This is probably due to the genetic influence of the mainland.
41:21We don't make rice paddies.
41:23It's an immune system.
41:25There are a lot of antibiotic cells in the blood,
41:28and when you get infected,
41:30there is a tendency for inflammation.
41:32It reacts to mold.
41:34Yes, it reacts to mold.
41:36This is just my imagination,
41:38but in the mainland,
41:40there is a possibility that the number of people has increased
41:43since agriculture started,
41:45and the number of infections has increased.
41:47There are a lot of people,
41:49and epidemics have been going on for a long time.
41:51That's right.
41:52I think that's why the number of mutations
41:54that are resistant to infections
41:56have accumulated in the genome.
41:59I see.
42:00It's connected to the cholera I mentioned earlier.
42:02When people get together,
42:03they get dirty,
42:04so in order to deal with that,
42:06they need to react as quickly as possible.
42:08That's right.
42:09I think that's why those people survived.
42:11Because they were naturally selected.
42:13Yes.
42:14What I want to say is that
42:16I don't want you to have the impression
42:18that you have a bad thing.
42:20On the contrary,
42:21everyone has it.
42:23And everyone has it for historical reasons.
42:26If you say,
42:27I have a bad mutation,
42:29so let's get rid of it,
42:30everyone will be gone.
42:31It's not like that.
42:32I think the important point is
42:34to make it possible to live
42:36while compensating for the genetic weakness
42:38that everyone has little by little.
42:40I see.
42:41I was surprised to find out
42:43that there was such a connection
42:45between the Chomon people and us now.
42:47It was 10,000 years ago.
42:49Of course,
42:50there have been many cases of illness,
42:52but that doesn't mean
42:53that it's all decided.
42:54Yes.
42:55You said,
42:56I have a bad mutation,
42:57I have a bad mutation,
42:58but it's not just that.
42:59I think it would be nice
43:00if we could understand that.
43:02By understanding,
43:03I think it would be nice
43:04if we could get rid of the idea
43:06that we don't know about that person
43:08until the end.
43:10I think it would be nice.